NIK as a Druggable Mediator of Tissue Injury: Trends in Molecular Medicine
NIK SMI1 | NF-κB Inhibitor | MedChemExpress
Nik‐related kinase is targeted for proteasomal degradation by the chaperone‐dependent ubiquitin ligase CHIP - Naito - 2020 - FEBS Letters - Wiley Online Library
NF-κB-Inducing Kinase Is Involved in the Activation of the CD28 Responsive Element through Phosphorylation of c-Rel and Regulation of Its Transactivating Activity | The Journal of Immunology
NF-κB-Inducing Kinase Is Involved in the Activation of the CD28 Responsive Element through Phosphorylation of c-Rel and Regulation of Its Transactivating Activity | The Journal of Immunology
Frontiers | Metabolic Reprogramming and Reactive Oxygen Species in T Cell Immunity | Immunology
MAP3K kinases and kidney injury - ScienceDirect
Structure of the Nuclear Factor κB-inducing Kinase (NIK) Kinase Domain Reveals a Constitutively Active Conformation - Journal of Biological Chemistry
PDF) NF- B-inducing kinase activates IKK- by phosphorylation of Ser-176
NIK as a Druggable Mediator of Tissue Injury: Trends in Molecular Medicine
NIK as a Druggable Mediator of Tissue Injury: Trends in Molecular Medicine
NIK SMI1 | ≥99%(HPLC) | Selleck | NF-κB inhibitor
NF-κB-Inducing Kinase Is Involved in the Activation of the CD28 Responsive Element through Phosphorylation of c-Rel and Regulation of Its Transactivating Activity | The Journal of Immunology
The Bruton tyrosine kinase inhibitor CC-292 shows activity in mantle cell lymphoma and synergizes with lenalidomide and NIK inhibitors depending on nuclear factor-κB mutational status | Haematologica
NIK as a Druggable Mediator of Tissue Injury: Trends in Molecular Medicine
NIK Antibody | Cell Signaling Technology
IJMS | Special Issue : NF-κB and Disease
Cells | Free Full-Text | Inhibitory-κB Kinase (IKK) α and Nuclear Factor-κB (NFκB)-Inducing Kinase (NIK) as Anti-Cancer Drug Targets | HTML
Activation of NF-B via a TLR2-MyD88-dependent TAK1-NIK-IKK/-IB pathway... | Download Scientific Diagram
NF-κB-inducing Kinase Phosphorylates and Blocks the Degradation of Down Syndrome Candidate Region 1* - Journal of Biological Chemistry